New Approach for Getting Better Bandwidth in Wireless Mobile Computing

Authors

  • Anirban Chakraborty M.Tech, Department of Artificial Intelligence, Lovely Professional University, Phagwara, Punjab, India, Author
  • Bhupinder Kaur M.Tech, Department of Computer Science & Engineering, Lovely Professional University, Phagwara, Punjab, India Author
  • Ruchika M.Tech, Department of Computer Science And Engineering, Lovely Professional University, Phagwara, Punjab, India Author

Keywords:

Mobile, Computing, Odessey, Rover, Agent, Sensor

Abstract

Wireless mobile computing is type of  computation by which various mobile gadgets gives  services to us at numerous ways. In wireless computing  of mobile one can find the working of sensors ad hoc  networks odysseys and rover etc. Not only has this  wireless based mobile computing service also suggested  that a user can take the services of networks any time  anywhere without any disturbances[1]. This includes the  physical location time place authentication etc. which  paves way for making mobile services successful. The  research paper also describes about merits and  disadvantages of wireless mobile computing along with  many challenges faced by it in remote areas.  Additionally, this paper also states how we get adapted to  the ecosystem of wireless network from the long process  of wired networks. Mobile agent is one of the most  important elements in computing. It is clearly described  in this research paper. This research paper also throws  ample amount of light on the working of sensors and its  unique features[12]. Wireless computing makes our life  totally comfortable by its outstanding services. Here all  useful services of that are clearly mentioned. At last the  conducting of wireless mobile computing in all areas of  the world is much more important for the welfare of future generations [9]. This research paper describes  about the steps with measures needed in implementing  wireless computing everywhere. 

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References

Alles, N., Soysa, N., Hayashi, J., Khan, M., Shimoda, A., Shimokawa, H., Ritzeler, O., Akiyoshi, K., Aoki, K., Ohya, K., 2010. Suppression of NF-kappaB increases bone formation and ameliorates osteopenia in ovariectomized mice. Endocrinology 151, 4626– 4634.

Almeida, M., O’Brien, C.A., 2013. Basic biology of skeletal aging: role of stress response pathways. J. Gerontol. A: Biol. Sci. Med. Sci. 68, 1197–1208.

Altindag, O., Erel, O., Soran, N., Celik, H., Selek, S., 2008. Total oxidative/anti-oxidative status and relation to bone mineral density in osteoporosis. Rheumatol. Int. 28, 317–321.

Baek, K.H., Oh, K.W., Lee, W.Y., Lee, S.S., Kim, M.K., Kwon, H.S., Rhee, E.J., Han, J.H., Song, K.H., Cha, B.Y., Lee, K.W., Kang, M.I., 2010. Association of oxidative stress with postmenopausal osteoporosis and the effects of hydrogen peroxide on osteoclast formation in human bone marrow cell cultures. Calcif. Tissue Int. 87, 226–235.

Bai, X.C., Lu, D., Bai, J., Zheng, H., Ke, Z.Y., Li, X.M., Luo, S.Q., 2004. Oxidative stress inhibits osteoblastic differentiation of bone cells by ERK and NF-kappaB. Biochem. Biophys. Res. Commun. 314, 197–207.

Burge, R., Dawson-Hughes, B., Solomon, D.H., Wong, J.B., King, A., Tosteson, A., 2007. Incidence and economic burden of osteoporosis-related fractures in the United States, 2005–2025. J. Bone Miner. Res. 22, 465–475.

Busino, L., Millman, S., Scotto, L., Kyratsous, C., Basrur, V., O’Connor, O., Hoffmann, A., Elenitoba Johnson, K., Pagano, M., 2012. Fbxw7alpha- and GSK3-mediated degradation of p100 is a pro survival mechanism in multiple myeloma. Nat. Cell Biol. 14, 375–385.

Cervellati, C., Bonaccorsi, G., Cremonini, E., Romani, A., Fila, E., Castaldini, M.C., Ferrazzini, S., Giganti, M., Massari, L., 2014. Oxidative stress and bone resorption interplay as a possible trigger for postmenopausal osteoporosis. Biomed. Res. Int. 2014, 569563.

Chang, J., Wang, Z., Tang, E., Fan, Z., McCauley, L., Franceschi, R., Guan, K., Krebsbach, P.H., Wang, C.Y., 2009. Inhibition of Osteoblast Functions by IKK/NF-jB in Osteoporosis. Nat. Med. 6, 682–689.

Guillerminet, F., Beaupied, H., Fabien-Soulé, V., Blais, A., 2010. Hydrolyzed collagen improves bone metabolism and biomechanical parameters in ovariectomized mice: an in vitro and in vivo study. Bone 46, 827–834.

Hamada, Y., Fujii, H., Fukagawa, M., 2009. Role of oxidative stress in diabetic bone disorder. Bone 45 (Suppl. 1), S35–S38.

Hendrickx, G., Boudin, E., Van Hul, W., 2015. A look behind the scenes: the risk and pathogenesis of primary osteoporosis. Nat. Rev. Rheumatol. 11, 462– 474Huang, Q., Gao, B., Jie, Q., Wei, B.Y., Fan, J., Zhang, H.Y., Zhang, J.K., Li, X.J., Shi, J., Luo, Zh.J., Yang, L., Liu, J., 2014. Ginsenoside-Rb2 displays anti-osteoporosis effects through reducing oxidative damage and bone-resorbing cytokines during osteogenesis. Bone 66, 306–314.

imi, E., Fukushima, H., 2016. NF-kappaB signaling pathways and the future perspectives of bone disease therapy using selective inhibitors of NF-kappaB. Clin. Calcium 26, 298–304.

Kadenbach, B., Ramzan, R., Vogt, S., 2009. Degenerative diseases, oxidative stress and cytochrome c oxidase function. Trends Mol. Med. 15, 139–147.

Kim, H.K., Kim, M.G., Leem, K.H., 2013. Osteogenic activity of collagen peptide via ERK/MAPK pathway mediated boosting of collagen synthesis and its therapeutic efficacy in osteoporotic bone by back-scattered electron imaging and microarchitecture analysis. Molecules 18, 15474– 15489.

Published

2020-06-04

How to Cite

New Approach for Getting Better Bandwidth in Wireless Mobile Computing . (2020). International Journal of Innovative Research in Computer Science & Technology, 8(4), 278–282. Retrieved from https://acspublisher.com/journals/index.php/ijircst/article/view/13228